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This model proposes an integrated framework for understanding the pathomechanisms of subclinical adrenal dysfunction, connecting deep physiological drivers to downstream clinical expressions. It serves as a working theory for practitioners who observe localized, non-systemic patterns of circadian dysrhythmia and persistent sympathetic dominance in their clients. This model demonstrates how a fundamental distortion in the balance of the nervous system can create a cascading series of effects, from cellular processes to the observable symptoms that manifest in the body. The simultaneous development of the sinew channels, the autonomic nervous system (ANS), the enteric nervous system (ENS), the limbic system, and the vestibule is why therapeutic work on the sinew channels can effect change across multiple systems. Because these signs of dysautonomia are visibly and objectively reflected in the fascial and sinew channel patterns, practitioners can learn to recognize the symptoms of localized cortisol inhibition and re-effect systemic balance through targeted interventions.

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